http://192.168.164.12:8010/index.php?title=EFNA3&feed=atom&action=historyEFNA3 - Revision history2024-03-29T07:13:18ZRevision history for this page on the wikiMediaWiki 1.30.0https://ngdc.cncb.ac.cn/lncrnawiki1/index.php?title=EFNA3&diff=1275014&oldid=prevFatima: Created page with "==Annotation== ===Name=== EFNA3 ===Alias=== NA ===Disease=== Disease: breast cancer [http://www.ncbi.nlm.nih.gov/pubmed/25023702 (PMID:25023702)] Dysfunction type: NA Desc..."2018-11-06T05:43:49Z<p>Created page with "==Annotation== ===Name=== EFNA3 ===Alias=== NA ===Disease=== Disease: breast cancer [http://www.ncbi.nlm.nih.gov/pubmed/25023702 (PMID:25023702)] Dysfunction type: NA Desc..."</p>
<p><b>New page</b></p><div>==Annotation==<br />
===Name===<br />
EFNA3<br />
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===Alias===<br />
NA<br />
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===Disease===<br />
Disease: breast cancer [http://www.ncbi.nlm.nih.gov/pubmed/25023702 (PMID:25023702)]<br />
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Dysfunction type: NA<br />
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Description: Sustained expression of both Ephrin-A3 and novel EFNA3 lncRNAs increased the metastatic potential of human breast cancer cells, possibly by increasing the ability of tumor cells to extravasate from the blood vessels into surrounding tissue. In agreement, we found a strong correlation between high EFNA3 expression and shorter metastasis-free survival in breast cancer patients. Hypoxia could contribute to metastatic spread of breast cancer via HIF-mediated induction ofEFNA3lncRNAs and subsequent Ephrin-A3 protein accumulation.<br />
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===Function===<br />
Function Mechanism: NA [http://www.ncbi.nlm.nih.gov/pubmed/25023702 (PMID:25023702)]<br />
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Biological Process: pathogenic process<br />
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Description: NA</div>Fatima